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In the TMLHE gene knockout mouse model, the carnitine biosynthetic pathway is inhibited due to the loss of TMLD activity in the liver, affecting the availability of carnitine in the heart, significantly reducing the rate of synthesis of LCACs, and contributing to the protection of mitochondrial function after myocardial IRI [215]

To determine the critical component responsible for increasing tear production, they utilized a mouse model of stress DED

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Y.et al (2016)

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That absence of evidence is not the same as evidence of no effect