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Western blotting Cell lysates were collected in RIPA buffer (150 mM NaCl, 1.0% NP-40, 0.5% sodium deoxycholate, 0.1% SDS (sodium dodecyl sulphate)and Western blotting was performed as previously described 49

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Classical experimental studies on reperfusion injury indicate that inflammation and oxidative stress are key determinants of secondary brain damage (Simon et al

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The molecular mechanisms proposed to explain how ROS could specifically mediate brain damage are the following: (1) The lower levels of antioxidants can promote increased activity of lipoxygenase in CNS stimulating leukotriene production, thereby increasing the immunoinflammatory processes in the cerebral cortex

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Animal models demonstrate increased fibroblast density and type I collagen expression in BPC-157-treated groups compared to controls. Sikiric, P

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