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Metastatic tumors have been shown to exhibit elevated levels of ROS, including HO, supporting the idea that a prodrug activated by intracellular HO could serve as an effective antimetastatic treatment

A preclinical study suggested its selective anticancer potential by depleting ROS below the levels required for telomerase activation, disrupting redox homeostasis, and suppressing cancer cell proliferation

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doi:10.1093/geronb/gbab194

The mechanism of these strains has demonstrated hypocholesterolemic effects through their ability to reduce serum levels of triglycerides, low-density lipoprotein (LDL), and high-density lipoprotein (HDL) (414)

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