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doi: 10.1677/joe.0.1680067 150 ZiebaDAAmstaldenMMortonSGallinoJLEdwardsJFHarmsPGet al

[Abstract]2023 Apr;62(4):464-478

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These findings provided new insight into the role of MRP6 in PXE, indicating that the disease could be a result of the absence of a substance that is normally excreted from the liver or kidney in the blood and is involved in tissue homeostasis[158]

3,41,42,43 To further explore the possible molecular mechanisms of DT-109, we analyzed the coronary transcriptome and demonstrated a significant DT-109-mediated reduction in the inflammatory response signaling pathway

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