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Epitope-specific mechanisms of IGF1R inhibition by ganitumab

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Targeted metabolomics was then used to confirm the expression of 4-ethylbenzoic acid (4-EA) levels and its role in CC was explored using cell counting kit-8, 5-ethynyl-2-deoxyuridine, colony formation, transwell, and wound healing assays

This insufficient targeting leads to low NADs concentrations at the disease site and unintended gene silencing or activation, potentially causing safety issues by requiring higher doses

Declarations The authors confirm that no paper mill and no artificial intelligence were used