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JAMA Pediatr 2013

Secondary carnitine deficiencies are most commonly the result of trapping of carnitine in fatty acylcarnitine due to defects in mitochondrial fatty acid -oxidation enzymes such as is the case in medium-chain acyl-CoA dehydrogenase deficiency (MCADD)

Additional experimental investigations are necessary to elucidate the relationship between human vascular disease and autophagy (183)

Curious about what L-carnitine really is

Las altas concentraciones de homocistena pueden promover el desarrollo de aterosclerosis (aterognesis) y trombognesis a travs de mecanismos que involucran estrs oxidativo y disfuncin endotelial, inflamacin, coagulacin sangunea anormal, y metabolismo lipdico desordenado (revisado en 35)