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As such, lipid-lowering agents have become a mainstay of therapy in the primary and secondary prevention of major adverse cardiovascular events (MACE) [28,29,30,31]

L-carnitine supplementation and physical exercise restore age-associated decline in some mitochondrial functions in the rat

[56] evaluating the association between TMAO and CVD events

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Water, Ascorbyl Palmitate, Urea, Butylene Glycol Dicaprylate/Dicaprate, Dimethicone, Acetyl Tyrosine, Dimethyl MEA, Fragrance, Zinc Sulfate, Sodium Hydroxide, Tocopheryl, Acetate, Panthenol, Sodium Hyaluronate, Ergothioneine, Pyridoxine HCl, Sodium Metabisulfite, Sodium Sulfite, Xanthan Gum, Disodium EDTA, Citric Acid, Famesol, Linalool, Amyl Cinnamal, Benzyl Salicylate, Cinnamal, Limonene, Eugenol, Geraniol, Hydroxycitronellal, Butylphenyl Methylpropional, Caprylyl Glycol, Ethylhexylglycerin, Hexylene Glycol, Phenoxyethanol

Lee BJ, Lin JS, Lin YC, Lin PT, Elsevier (2015) Antiinflammatory effects of L-Carnitine supplementation (1000 mg/d) in coronary artery disease patients