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Moreover, miR-483-3p is identified as a critical regulator of IGF-1 expression in natural killer cells
GlyT1 inhibition increases extracellular glycine availability at synaptic sites, thereby facilitating co-agonist occupancy at NMDA receptors and modulating glutamatergic neurotransmission
The goal of our study was to determine changes in brain GSH levels upon sulforaphane treatment, possibly in correlation with levels of blood GSH
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(PubMed) Zanzonico PB, Becker DV
