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[273] documented that the administration of FMT therapy has led to a notable enhancement in the capability of individuals with metabolic syndrome to respond to insulin [273]

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In line with this, SOX2 silencing abrogates the capability of cells with TP53 and RB1 loss to develop lineage plasticity and to express neuroendocrine markers [45]

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Furthermore, to evaluate the impact of the functional form of the CFR variable, we conducted a sensitivity analysis in which CFR was treated as a continuous variable (numerically reflecting increasing carbohydrate content), rather than as an ordinal variable