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Available to qualified researchers, laboratories, and institutions in Perth and across Western Australia, with Australian-based fulfilment

Mechanistically, GHK-Cu preserved mitochondrial function by increasing mitochondrial membrane potential, alleviating age-related mitochondrial network fragmentation, shifting mitochondrial dynamics toward fusion via regulating drp-1 and fzo-1 expression, and promoting ATP biosynthesis

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In muscle cells, MOTS-c markedly improves insulin signaling and metabolic function

Endothelial cells, which line the inside of blood vessels, play a key role in regulating blood pressure, blood flow, and clotting and they too appear to be positively affected by MOTS-c