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As expected, elevations in FABP5 S-glutathionylation of lung tissue induced by LPS were more pronounced in Grx1 fl/fl LysM cre mice (Fig

Additional studies to investigate the interaction between HNF4 and CBS will therefore help to better understand the complex role of CBS in tumor biology

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Thus, TLR2/TLR4 activation promotes the dissociation of the inhibitor of nuclear factor kappa-B (I-B)/NF-B complex through a myeloid differentiation primary response 88-dependent (MyD88) signaling pathway, mediating the nuclear translocation of nuclear factor kappaB (NF-B) and specifically acting on the NLRP3 and IL-1B gene promoter regions

J Med Internet Res 2023;25:e42090

doi: 10.1096/fj.01-0944rev