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Following hCiPSC treatment, the microbial community composition shifted towards the control state, as evidenced by significantly reduced dissimilarity to the VPA-ASD model (Figure S4B)

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[80] reported that 4-HNE could upregulate cyclooxygenase-2 (COX-2) via activating ATF/CRE and inhibit inducible nitric oxide synthase (iNOS) via inactivating NF-B in OA chondrocytes

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