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Macrophage studies demonstrate that GHK-Cu administration shifts these cells from a pro-inflammatory M1 phenotype to a tissue-repairing M2 phenotype, increasing their production of growth factors including vascular endothelial growth factor (VEGF) and platelet-derived growth factor (PDGF) by 40-60%

As such, it regulates critical cellular processes such as metabolism of estrogens, prostaglandins, leukotrienes, and xenobiotic drugs

This creates an immediate antioxidant surge in the blood, but not necessarily inside the cells where glutathione is actually required to function

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Two-tailed paired or unpaired Students t-test was applied