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Aim This review aims to provide an integrated synthesis that bridges mechanistic evidence (anti-inflammatory, antioxidant) with clinical outcomes (mortality, arrhythmias) for LC supplementation in CAD, while critically appraising inconsistencies across the literature (e.g., heart failure, reinfarction)

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However, ultrastructural analysis revealed that all mutant mice, including the 2aa, exhibited the same myofiber abnormalities as seen in the Alix / and Ozz / models (Supplementary Fig

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