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Results Senescence-associated markers were enhanced in keloid fibroblasts and lesions Analysis of the heterogeneity of keloid cells revealed 28 cellular clusters (027) using unsupervised clustering of 34,043 cells based on established lineage-specific marker genes (Supplementary Fig

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Angiogenesis and Blood Flow: GHK-Cu promotes vascular endothelial growth factor (VEGF) expression, stimulating new blood vessel formation

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Table 4)

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Previous studies utilizing SREBP-1 and SREBP-2 single-deficient mice suggest a potential compensatory mechanism in which the loss of one isoform may compensate for the loss of the other isoform [91, 92]

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