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Finally, including additional biomarkers in blood and urine could help to detect more subtle differences in oxidative damage in generally healthy subjects
JNK Depletion of GSH by NAPQI results in increased H 2 O 2 release from mitochondria to cytoplasm, which could oxidize thioredoxin and cause thioredoxin to disassociate from apoptosis signaling-regulating kinase 1 (ASK-1) [41]
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