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It is not unlikely that hyperglycemia-stimulated mitochondrial dysfunction and apoptosis account for cardiac metabolic memory [200]
When stress becomes chronic, it can lead to persistent activation of the HPA axis, causing consistently high levels of cortisol
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mtDNA: mitochondrial DNA
Exploring the deep-seated connections between dysbiosis and PD could thus open new avenues for PD treatment
Investigating these genes in greater detail allows researchers to deepen their understanding of the genetic architecture of depression and to identify potential biomarkers for personalized treatment strategies
