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and Uchio, E

ASAIO J

Licochalcones B, C, D and H induced apoptosis in OSCC cells via different mechanisms including ROS generation, inhibition of the JAK2/STAT3 signaling pathway with downregulation of genes such as surviving, p21 and p27, or via regulation of matrin 3, a protein involved in several cell processes such as apoptosis, DNA replication, transcription and translation [141,142,143,144,145]

The adverse effects seen were attributed to the occurrence of apoptosis, as there was an increase in caspase 3 activity and a decrease in Bcl-2 activity

Semax has been shown in some studies to influence neurotrophic factors such as BDNF upregulation and other pathways involved in neuroprotection and cognitive processes