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Because reactive oxygen species (ROS) have been implicated as mediators of inflammatory bowel disease (IBD), the purpose of the present work was to determine the functional role of mucosal GSH in the trinitrobenzenesulfonic acid in 50% ethanol (TNBS+ethanol)-induced colitis in rats

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Numerous cells, including erythrocytes, hepatocytes, and cardiomyocytes, contain the thiol tripeptide glutathione (GSH) [1], which is made up of 3 amino acids, l-glutamyl, l-cysteinyl, and glycine [2].

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