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doi: 10.1016/j.jpba.2006.04.002 79 HordieiKGontovaTTrumbeckaiteSYaremenkoMRaudoneL

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Lutchmansingh et al.s findings indicate that such deficiencies are due to decreased synthesis and higher non-glycaemic utilisation in patients with microvascular complications [2]

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that correcting vitamin D deficiency can, in itself, increase GSH levels

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oxidative stress (OS)

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It then inhibits adipocyte lipolysis and increases FFAs in cells, further stimulating the LDLs accumulation and secretion, inducing lipid metabolism disorders, lipotoxicity, and even cell necrosis, finally resulting in diabetic myocardial damage

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10.3389/fimmu.2022.833355 Front Immunol

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