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Kerins MJ, Ooi A

Finally, many exhibit anti-fibrotic and cardiomyocyte-protective actions: they interfere with fibrosis-related pathways (TGF-/Smad, etc.), inhibit cardiac fibroblast activation, and reduce pathological deposition of extracellular matrix in the heart (Xiao et al., 2018

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(2005) Band 3 modifications in Plasmodium falciparum -infected AA and CC erythrocytes assayed by autocorrelation analysis using quantum dots

Relations between polymorphisms in drug-metabolising enzymes and toxicity of chemotherapy with cyclophosphamide, thiotepa and carboplatin

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