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& Alenghat, T

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In diabetic podocytes, hyperglycemia-induced MAMs hyperplasia exacerbates mitochondrial dysfunction and ER stress, promoting podocyte apoptosis and DAMP release

This enzyme catalyzes the production of fructose-2,6-bisphosphate, an activator of phosphofructokinase-1 (PFK), which, in turn, reduces the efficiency of glycolysis in neurons

Victoria Martin M, Fernando Fiol D, Sundaresan V, Julin Zabaleta E, Pagnussat GC

GLP-1 receptor agonists may reduce symptoms through anti-inflammatory and metabolic mechanisms, but they do not eliminate endometrial implants or cure the underlying condition