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Cell death in these experiments was suppressed by co-treatment with the specific ferroptosis inhibitor ferrostatin-1, confirming that p53 stabilization did not alter the mode of cell death induced by erastin2 (Fig 1C)

7) and NMR method for determining antioxidant polyphenolic acids in the medicinal herb

CPP-CAND disrupts FOXO4-p53 foci and induces apoptosis through caspase-dependent pathways FOXO4 is progressively recruited to euchromatin foci following senescence induction and is retained in the nucleus of senescent cells through its interaction with p53, contributing to the maintenance of the senescent state 34

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By managing oxidative stress in the dermal layers, it helps maintain the skin's natural glow and resilience against environmental stressors

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