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Considering these previous observations, we hypothesized that CoQ deficiency might induce sublethal effects, leading to an adaptive reconfiguration of the mitochondrial network

Application of FMOs Inhibitors FMOs are monooxygenase that oxygenates nucleophilic heteroatom-containing chemicals and drugs and converts them into harmless, polar metabolites that are easily excreted (Krueger and Williams, 2005)

Sagorijevanjem masti u mitohondrijima oslobaa se energija, to moe poveati ukupnu razinu energije

doi: 10.1038/oncsis.2014.18 52 Audet-WalshEDufourCRYeeTZouanatFZYanMKalloghlianGet al

For instance, treatment with TMAO was found to lower the capacity of human umbilical vein endothelial cells (HUVECs) compared to the control group, suggesting that TMAO can induce vascular endothelial dysfunction through the activation of the NF-B pathway (Ma et al., 2017)

[Google Scholar] Shalev DP, Soffer Y, Lewin LM.1986