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Direct Evidence for A Role of Redox Dysregulation in Als In this section, we discuss evidence demonstrating that the central cellular redox regulatory mechanisms and associated proteins are perturbed in ALS (Table 2)

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When studying the function of the subcapsular sinus in LNs, we unexpectedly observed that intact lymph-borne IgG antibodies had instant access to the parenchyma of draining LNs in a process independent of reticular conduits

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The BH3-only protein is considered a sensor of the mitochondrial apoptosis pathway, and it is the upstream component of the Bcl-2 family to regulate mitochondrial apoptosis [83]