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Description
Mutations in, or RNAi depletion of, unc-116 (kinesin heavy chain) and klc-1 (kinesin light chain) both resulted in an abnormal, uniform distribution of both cLDs and ER (Fig 12A and 12B), and disrupted the normal, asymmetric localization of soluble ER residents within germ cells (inset, Fig 12B)

The conserved His-D-Phe-Arg-Trp motif is the melanocortin active site responsible for receptor engagement

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Migraine and neuroinflammation: the inflammasome perspective

A Novel Brain-Expressed Protein Related to Carnitine Palmitoyltransferase I
Rupp G'16 Nicholas R
