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Research has shown that BAs in combination with its activating transcription factor, farnesoid X receptor (FXR), inhibit cholesterol transport and increase cholesterol excretion by macrophages in order to reduce cholesterol concentration in plasma, and can be used as a therapeutic target for dyslipidemia (Figure 2

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7.4 Multisystem effects Beyond the cardiovascular and hepatic axes, chronic exposure to anabolicandrogenic steroids (AAS) has been associated with adverse effects across multiple additional organ systems