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Mitochondrial dysfunction is a fundamental and complex hallmark in many neurodegenerative disorders, including PD

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Studies have shown that muscle RING-type E3 ubiquitin ligase 1 (MuRF1, TRIM63) and muscle atrophy F-box protein (MaFbx) in the ubiquitinproteasome pathway are closely related to the degradation of muscle protein in cachexia patients [15,16,17]

An alternative therapeutic approach that has been developed on the basis of this problem is the use of arginase-1 peptide vaccines that activate T cells to target and recognize cells expressing arginase 1

doi: 10.3390/nu13041238