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Hyperglycemia drives oxidative stress through several pathways, including the polyol and hexosamine pathways and the formation of advanced glycation end products (AGEs) [3]

doi: 10.1016/j.cld.2018.01.007 [PubMed] 30777470 Hendrickson RG

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3.1.3 Primary defects of the synthesis or activity of IGF-1 and IGF-2 This category of defects includes defects of the synthesis of IGF-1 and IGF-2 due to gene mutation, abnormalities of the IGF-1 receptor, bio inactive IGF-1, and defects of IGFBP-3 or ALS