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Excess fat and ROS damage cells, causing inflammation 69, 70

Biochemically, GGT is associated directly with GSH regulation and maintenance of its normal redox status in cells by breaking down extracellular GSH which cannot cross the cellular membrane to release cysteine and glycine building blocks which can more easily cross into the cell for de-novo synthesis of GSH during periods of oxidative stress [26]

Blockade of the N-methyl-D-aspartate glutamate receptor ameliorates lipopolysaccharide-induced renal insufficiency

Although the intake of antioxidants minimizes complications related to diabetes, the mechanism behind the antioxidant action requires further studies (Rajendiran et al

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