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Schotman, H., Karhinen, L

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In high-glucose-treated HK-2 tubular cells, MitoQ upregulated Nrf2 translocation, inhibited Kelch-like ECH-associated protein 1 (Keap1), and blocked M1 macrophage polarization and Th17 cell infiltration through mitochondrial quality control mechanisms, establishing a link between mitochondrial redox balance and immune cell dysregulation in DKD (126, 127)

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By releasing mtDNA and triggering cGASSTING activation, mitochondrial dysfunction acts as a key upstream driver of neuroimmune responses in HD