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The pathomechanisms of these diseases are multifactorial (Fig
It is now clear that oxidative damage, including that associated with small A42 oligomers, by targeting many aspects of brain cell structure and function, is fundamental to the pathogenesis and progression of AD
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Previous research results show that antioxidant activity of LAB strains could be attributed to their production of cell-surface active compounds including proteins, lipoteichoic acid or extracellular polysaccharides (Li et al., 2012
