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These resistant cells upregulate lipid ROS-detoxifying enzymes such as GPX4 and FSP1, and knockdown of GPX4 in these resistant cells re-sensitizes them to both ferroptosis and TMZ [155], highlighting a glioblastoma-specific therapeutic vulnerability that emerges from alkylating agent adaptation

Linkage of aerobic glycolysis to sodium-potassium transport in rat skeletal muscle

Telomeres, which protect chromosomal ends from degradation, are repaired less efficiently than the rest of the genome [118]

A.ZhouH.LiX

Excess iron could promote the Fenton reaction in PD lesions, that is, lead to the production of more ROS, which is unfavorable for the treatment of PD

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