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Abstract Changes in the composition of gut microbiota have been associated with the development of Alzheimers disease (AD), and diverse primary mechanisms are proposed

Emodin-induced autophagy promoted Snail and -catenin protein degradation, inhibiting EMT partly through the PI3K/AKT/mTOR and Wnt/ -catenin pathways

Individuals with inherited defects in selenocysteine insertion sequence (SECIS)-binding protein 2 display a syndrome of selenoprotein-related defects including abnormal thyroid hormone metabolism, delayed bone maturation, and other more individual phenotypes

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Most patients feel fine immediately after treatment and resume activities