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Genomics 57 , 256267 (1999)
By decreasing the level of intracellular glutathione with l-buthionine-sulfoxamine (BSO) a specific inhibitor of glutathione synthesis, the cytotoxic effect of gliotoxin was significantly attenuated
NMNH is dephosphorylated into NRH to be transported into the cell through equilibrative nucleoside transporters (ENTs) 35 , where it is phosphorylated back into NMNH by adenosine kinase (AK) 36
(4) 1-ns NPT simulation with t = 2 fs)
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The logic behind post-surgical use mirrors general injury recovery
