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Notably, researchers observed that the rise in p21 levels, a marker of senescence, was independent of p53, indicating that, unlike many previous drugs, AKI603-induced senescence may operate independently of p53 activity [79]
As reperfusion therapy [145], there was a combined effect on particularly vulnerable hippocampal neurons [236], counteracting both early and delayed neural damage [145]
DOI: 10.1371/journal.pone.0130189 58
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