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Yu T, Gao M, Yang P, Liu D, Wang D, Song F, Zhang X, Liu Y

In such cases, TSH receptor signaling becomes dysregulated, often due to TSH receptor-stimulating antibodies or somatic mutations in thyroid follicular cells, leading to increased thyroid hormone output despite suppressed TSH levels [26]

[DOI] [PubMed] [Google Scholar] 360.Solfrizzi V., Panza F., Imbimbo B.P., D'Introno A., Galluzzo L., Gandin C., et al

Electroencephalogr Clin Neurophysiol 66 : 7581

While Figures 1, 2 summarize ASTs modulation of oxidative and inflammatory pathways, it is important to note that these models are derived largely from preclinical studies

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