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Over-the-counter anti-oxidant therapies for use in multiple sclerosis: a systematic review
When mitochondria are damaged, PINK1 first accumulates on the mitochondrial outer membrane and then recruits Parkin, which is considered to be required for initiating mitochondrial autophagy in vitro models, to initiate the mitochondrial autophagy

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MET acts as a substrate for several protein tyrosine phosphatases, including the receptor PTPs density enhanced phosphatase 1 (DEP 1) and leukocyte common antigen related (LAR) and the non-receptor PTPs, PTP1B and T cell PTP [32]

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