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Its main active ingredient, total dajizin triterpenes, reduces the expression of ACSL4 in the rat model of RA, increases the expression of glutathione and GPX4, and upregulates Kelch-like ECH-associated protein 1 and HO-1 in the Nrf2/HO-1/GPX4 pathway, indicating that total dajizin triterpenes can inhibit cell aberrant ferroptosis by suppressing lipid peroxidation and thus exert therapeutic anti-RA effects [168]

FLG mutations are linked to a history of AD onset, disease severity, and disease persistence

Use of a scaffold peptide in the biosynthesis of amino acid-derived natural products

Reagents and general procedures from the Gibson assembly cloning kit from New England Biolabs (NEB) were used

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