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Heightened PKC activity in hypoxia/ischemia-reperfusion appears to promote inflammation and oxidative stress at the subcellular level

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These are known as "ketone bodies" and the process as "ketogenesis", and they are readily transported to extrahepatic tissues and converted back to acetyl-CoA, which then enters the citric acid cycle and is oxidized in mitochondria for energy (ATP production)

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In contrast, lacosamide blocks the slow inactivation state, which may explain the added benefit of lacosamide in combination with carbamazepine

Zhang CX, Ho SC