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As GABA and glutamate hold opposite effects modulating the neural activity (either decreasing or increasing excitability), it may be possible that the relation of these neurometabolites with GSH derives from the energy supply needed to hold either inhibitory or excitatory connections and not from the energy of neural activity itself

Transgenic mice expressing human PON3 have been shown to be protected from the development of atherosclerosis, without any significant changes in plasma lipoprotein cholesterol, triglyceride or glucose levels

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