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Exogenous AAS inhibit endogenous testosterone production, and discontinuation of their use is often associated with a prolonged decrease in testosterone secretion (Kanayama et al., 2015

They found the binding sites of NAM and SAH in NNMT were adjacent to each other and D197 and Y20 in NNMT protein are indispensable for NAM methylation [18]

& Nonogaki, K

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This is one of the core pathological mechanisms underlying obesity, diabetes, and other metabolic diseases