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[DOI] [PubMed] [Google Scholar] Mossanen J

These findings suggest that functional polymorphisms influencing inflammation and/or oxidative stress may serve as heritable markers associated with other allelic variants predisposing individuals to oxidative stress and inflammation (Rabelo et al., 2018)

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Conversely, excessive SAM accumulation without sufficient GSH conversion may promote ROS generation and activate the NLRP3 inflammasome, leading to IL-1 secretion (67)

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