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Utilizing GC/MS, we measured other markers that could be altered by exposure to mycotoxins

So thats 2 CTs and 2 MRIs at this point, 3 of those (minus the possible thrombosis one) are CLEAR

N-acetyl-L-cysteine (NAC), a well-characterized thiol antioxidant and precursor of reduced glutathione (GSH), not only scavenges intracellular ROS directly but also replenishes depleted GSH stores in erythrocytes, thereby inhibiting ROS-mediated Ca 2+ influx and PS externalization induced by eryptosis triggers such as desipramine or uremic toxins

However, metformin inhibits mitochondrial enzymes to activate AMPK, and the effects on cell protection could be AMPK- or autophagy-dependent and autophagy-independent [249, 250]

Marienne is likable
