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Description
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Zarrindast and Khakpai, 2015), and both D1- and D2-like receptors, as well as DAT, can mediate these effects (de la Mora et al., 2010

the programmable half-life platform introduced in this work may provide an additional level of control to obtain maximum efficacy with minimal adverse effects when moving into clinical trials

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Recent research indicates that Hcy disrupts the breakdown of fats in fat cells by activation of the AMP-activated protein kinase (AMPK) pathway (Wang et al., 2011)

Japanese Journal of Psychiatry and Neurology
