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To determine whether the lipid-loss phenotype of pqm-1 mutants is primarily caused by a downregulation of fatty acid synthesis through decreased fat-7 expression, we performed a rescue experiment by re-expressing fat-7 under its own promoter ( fat-7p::fat-7::gfp translational reporter) and measuring lipid levels

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Automated data acquisition was performed using EPU v 3.4, with a defocus range of 1 to 1.5 m for both OCTN2-inward and OCTN2-ipratropium, and 0.5 to 2.0 m for OCTN2-carnitine

Glycation affects nucleic acids, leading to mutations, and disease like diabetes and cancer