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All tumour regions had ATP/ADP and PCr/ATP ratios comparable to those in normal brain (Fig

However, in the second reaction, the conversion and yield were significantly reduced, and the reaction was incomplete after 48 h

Finally, we highlight the potential for SIRT1-based therapeutics for inflammatory diseases

10.1016/j.tox.2019.05.009 91 XingW

Activated p53 triggers cell cycle arrest to provide damaged DNA with enough time to repair or, in the case of excessive damage, to initiate apoptosis, thus preventing the proliferation of cells carrying severe genetic defects (42, 43)

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