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Principally, this is due to the fact that individuals who are highly B vitamin deficient may experience a delay in the conversion of Cyanocobalamin into Methylcobalamin

Structural basis for assembly and disassembly of the IGF/IGFBP/ALS ternary complex

Metabolic Decompensation after VitB12 Deficiency The deficiency of VitB12, or more specifically, the alterations in MTR or MUT (for which VitB12 serves as a cofactor), not only avoids the biosynthesis of reaction intermediates but also determines the accumulation of substrates that are toxic at higher concentrations (Figure 2)

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Suchting R, et al

Also, GSH facilitates the transport and excretion of toxins, through the formation of S-conjugates of activated intermediates, which are water soluble and undergo renal excretion