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Description
AMPK supported IDH2-catalyzed mitochondrial NADPH regeneration to maintain redox homeostasis in a FAO-dependent manner The BRB injury was primarily contributed by the endothelial mitochondrial ROS, with FAO being the main source of endothelial mitochondrial respiration rather than glycolysis [28]
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doi:10.1186/1471-2407-14-378
& Kalda, A
