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Under neuroinflammatory conditions, dysfunctional mitochondria release ROS and damage-associated molecular patterns (DAMPs) (53), activating microglia and astrocytes, and triggering the release of pro-inflammatory cytokines and chemokines (54), thereby exacerbating the damage

Jiang, J.-S

This signaling, even when mediated through the same pathways, can have different spatial and temporal localization and is associated with distinct consequences [106]

Glucose-6-phosphate isomerase is associated with disease activity and declines in response to infliximab treatment in rheumatoid arthritis

It remains questionable as to whether similar effects would be seen in humans at moderate, reasonable doses

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